Reports on adenomyosis in pregnancy are few and include a small number of patients. Currently, ultrasound is the first-line imaging diagnostic tool, with a sensitivity of about 84%. Controversies exist regarding the relation of adenomyosis with infertility and pregnancy complications. We analyzed the intrauterine growth restriction (IUGR) cases for a period of one year aiming to determine if adenomyosis impacts negatively the pregnancy outcome. The preterm birth rate in our study population was 20%. From the preterm birth subgroup, the greatest percentage was attributed to late prematurity. The incidence of adenomyosis in this group of pregnancies complicated with IUGR was about 9%. According to our results, there is a higher incidence of preterm birth incases complicated with fetal growth restriction, as reported by other much larger studies. At this point, there is no established associated risk or method to prevent unfavorable outcomes related to the impact of adenomyosis on pregnancy.

Fetal growth restriction (FGR) is a complication diagnosed in about 10% of pregnancies and is associated with significant perinatal mortality and morbidity. Both the diagnosis and the correct management of cases of intrauterine growth restriction remain a challenge of modern obstetrics. A normal placental development is essential for a proper intrauterine physical and neurological growth of the fetus throughout pregnancy. Various pathophysiological situations may reflect in abnormal placental development linked with severe pregnancy disorders. In this paper we aim to exemplify sonographic aspects in various placental pathology associated with FGR along with the recommended management. Placental insufficiency is the most common risk factor for FGR and it cannot be directly measured and objectified, remaining a diagnostic of exclusion. The risk for perinatal adverse outcomes in placenta accreta spectrum cases is increased trough the pathological implantation especially in depth. In pregnancies complicated with placental insufficiency, secondary macroscopic lesions can be noted, as parabasal and intervillous thrombosis, hematomas, extensive fibrin deposits and infarcts areas. Even if, over time, multiple studies have targeted methods of preventing intrauterine growth restriction through actions on the mother, the effectiveness of no treatment has been demonstrated.